By Linda S. Costanzo PhD
- NEW! Full-color layout, move charts, illustrations, and tables that summarize info for handy review
- Each bankruptcy is written within the renowned Board assessment Series (BRS) define layout and lines bolded key words to streamline your study
- Over 350 USMLE-style questions, solutions, and rationales either electronically and in print toughen your body structure review
- A FREE spouse site deals a web book and an interactive query financial institution with the entire questions from the e-book so that you can customise your overview tests!
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Additional info for BRS Physiology
The cross-bridge cycle begins (see Figure 1-12): a. At first, no ATP is bound to myosin (A), and myosin is tightly attached to actin. In rapidly contracting muscle, this stage is brief. , rigor). b. ATP then binds to myosin (B), producing a conformational change in myosin that causes myosin to be released from actin. c. Myosin is displaced toward the plus end of actin. There is hydrolysis of ATP to ADP and inorganic phosphate (Pi). ADP remains attached to myosin (C). d. Myosin attaches to a new site on actin, which constitutes the power (force-generating) stroke (D).
These sites are innervated by nerves that arise from the same segment of the spinal cord. For example, ischemic heart pain is referred to the chest and shoulder. C. Vision 1. Optics a. Refractive power of a lens ■ ■ ■ is measured in diopters. equals the reciprocal of the focal distance in meters. Example: 10 diopters = 1/10 meters = 10 cm b. Refractive errors (1) Emmetropia—normal. Light focuses on the retina. (2) Hypertropia—farsighted. Light focuses behind the retina and is corrected with a convex lens.
Adrenergic receptors (adrenoreceptors) a. `1 Receptors ■ ■ ■ ■ are located on vascular smooth muscle of the skin and splanchnic regions, the gastrointestinal (GI) and bladder sphincters, and the radial muscle of the iris. , contraction or constriction). are equally sensitive to norepinephrine and epinephrine. However, only norepinephrine released from adrenergic neurons is present in high enough concentrations to activate α1 receptors. Mechanism of action: Gq protein, stimulation of phospholipase C, and increase in inositol 1,4,5-triphosphate (IP3) and intracellular [Ca2+].