By R. A. Freedland

Though the most important emphasis of this booklet can be references to numerous easy texts are given on the to supply the nutritionist with a biochemical finish of the creation. method of his experimental and useful To facilitate effortless reference, the booklet has difficulties, it truly is was hoping that the publication can also be been divided into chapters in line with the of use to the biochemist and physiologist to roles of the fundamental foodstuff in metabolism. display how nutritional meals manipula­ inside chapters, dialogue will comprise such tion can be utilized as a strong software in fixing subject matters because the results of food on metabolism, difficulties in either body structure and biochemistry. the destiny of nutrien ts, the jobs of varied tissues there'll be no try and write an all-encom­ and interplay of tissues in using foodstuff, passing treatise at the courting among and the biochernical mechanisms concerned. biochemistry and nutrients; quite, it's was hoping towards the tip of the publication, a number of instance that the feedback and partial solutions provided difficulties could be awarded, which we are hoping will right here will give you the reader with a foundation for give you the reader with the chance to imminent difficulties and designing experi­ shape testable hypotheses and layout experi­ ments.

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F. and Lowenstein, J. M. (1962),J. biol. , 237,3640. , Abraham, S. and Chaikoff, I. L. (1965), Biochern. Biophys. Res. , 19, 770. [14] Martin, D. B. and Vagelos, P. R. (1962), J. biol. , 237,1787. [15] Rognstad, R. and Katz, J. (1966),Proc. natn. Acad. , (USA), 55, 1148. , Ringelmann, E. and Lynn, F. (1965), Biochern. , 343,243. [17] Veech, R. , Eggleston, L. V. and Krebs, H. A. (1969),Biochern. ,115,609. [18] Katz, J. and Wals, P. A. (1971), Arch. biochern. , 147,405. [19] Sullivan, A. , Miller, N.

The form of dietary carbohydrate also influences the lipogenic enzym€ls. When sucrose replaces dietary glucose or starch, there is a marked increase in the enzymes associated with 32 lipogenesis, particularly in liver [6] . In addition, the fructose moiety, ente ring glycolysis beyond the regulation point at phosphofructokinase, can provide increased substrate for the liver to carry on lipogenesis. This increased potential for lipogenic activity in liver may be more detrimental than a comparable increase in adipose tissue, since the liver is the primary site of cholesterol synthesis.

Shifts in the balance of these processes are generally to the animal's benefit, allowing adaptation to the situation (Fig. 5). One shift that appears to be to the detriment of the animal, however, occurs with the consumption of alcohol. In alcoholics and even in 'social drinkers' there is increased lipid deposition in the liver. Whether this fatty liver syndrome is the cause of cirrhosis of the liver is controversial, but the one is often followed by the other. 7 Effects of alcohol Fatty liver in alcoholics was formerly thought to be a consequence of inadquate dietary protein intake and the resultant shortage of proteins for transporting lipid from the liver to the periphery.

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